diabetes

[dahy-uh-bee-tis, -teez]

noun

Pathology.

1. any of several disorders characterized by increased urine production.

2. Also called diabetes mellitus. a disorder of carbohydrate metabolism, usually occurring in genetically predisposed individuals, characterized by inadequate production or utilization of insulin and resulting in excessive amounts of glucose in the blood and urine, excessive thirst, weight loss, and in some cases progressive destruction of small blood vessels leading to such complications as infections and gangrene of the limbs or blindness.

3. type 1 diabetes.

4. type 2 diabetes.

5. Also called diabetes insipidus. increased urine production caused by inadequate secretion of vasopressin by the pituitary gland.

diabetes

/ -tiːz, ˌdaɪəˈbiːtɪs /

noun

1. any of various disorders, esp diabetes mellitus, characterized by excretion of an abnormally large amount of urine

Word History and Origins

Origin of diabetes¹

First recorded in 1555–65; from New Latin, Latin, from Greek, equivalent to diabē- (variant stem of diabaínein “to go through, pass over,” equivalent to dia- “through” + baínein “to pass”) + -tēs agent suffix; dia-

Word History and Origins

Origin of diabetes¹

C16: from Latin: siphon, from Greek, literally: a passing through (referring to the excessive urination), from diabainein to pass through, cross over; see _diabase

Example Sentences

Professor and endocrinologist Simone van de Sande Lee is clear: "These are drugs approved to treat conditions like diabetes or obesity - they are not cosmetic tools."

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"Problems with mitochondria and inflammation linked to damaged mtDNA have been connected to diseases such as neurodegeneration and diabetes," he said.

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Although obesity is widely known to increase the risk of chronic conditions such as heart disease and type 2 diabetes, this study suggests its influence on the brain may develop much earlier.

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Diets high in ultraprocessed foods are linked to obesity, Type 2 diabetes, cardiovascular disease and cancer.

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In that earlier work, the researchers first triggered diabetes in mice by using toxins to destroy the insulin-producing cells in the pancreas.

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