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Creutzfeldt-Jakob disease

or Creutz·feldt-Ja·cob disease

[kroits-felt yah-kawp]
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noun Pathology.
  1. a rare, usually fatal brain disorder thought to be caused by an infectious prion and characterized by progressive dementia, blindness, and involuntary movements.
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Origin of Creutzfeldt-Jakob disease

1965–70; after German physicians Hans G. Creutzfeldt (1885–1964) and Alfons Jakob (1884–1931)
Dictionary.com Unabridged Based on the Random House Unabridged Dictionary, © Random House, Inc. 2018

British Dictionary definitions for creutzfeldt-jakob disease

Creutzfeldt-Jakob disease

noun
  1. pathol a fatal slow-developing disease that affects the central nervous system, characterized by mental deterioration and loss of coordination of the limbs. It is thought to be caused by an abnormal prion protein in the brain
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Word Origin

C20: named after Hans G. Creutzfeldt (1885–1964) and Alfons Jakob (1884–1931), German physicians
Collins English Dictionary - Complete & Unabridged 2012 Digital Edition © William Collins Sons & Co. Ltd. 1979, 1986 © HarperCollins Publishers 1998, 2000, 2003, 2005, 2006, 2007, 2009, 2012

creutzfeldt-jakob disease in Medicine

Creutzfeldt-Jakob disease

(kroitsfĕlt-)
n.
  1. A rare, usually fatal encephalopathy that is likely caused by a prion and is characterized by progressive dementia and gradual loss of muscle control, usually in middle age.Jakob-Creutzfeldt disease
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The American Heritage® Stedman's Medical Dictionary Copyright © 2002, 2001, 1995 by Houghton Mifflin Company. Published by Houghton Mifflin Company.

creutzfeldt-jakob disease in Science

Creutzfeldt-Jakob disease

[kroitsfĕlt-yäkôp]
  1. A rare, usually fatal encephalopathy that occurs most often in middle age and is likely caused by a prion. It is characterized by progressive dementia and gradual loss of muscle control. The disease is named after its discoverers, German pathologist Hans Gerhard Creutzfeldt (1885-1964) and German neurologist Alfons Maria Jakob (1884-1931).
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The American Heritage® Science Dictionary Copyright © 2011. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.